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What are Cardiac Defibrillators and ICDs?
Cardiac or heart defibrillators are used to restore a normal heart rate in patients who have ventricular tachycardia or ventricular fibrillation.
In a normally functioning heart, the heart beat or rate is controlled by small group of cells called the 'sinus node' near the filling chambers of the heart, called the atria. The sinus nodes generates an electrical impulse that flows from the atria or filling chambers to the pumping chambers or ventricles.
When a person has damage to the heart muscles, from a heart attack, heart inflammation (cardiomyositis), enlarged heart (cardiac hypertrophy) or damaged heart valves, the ventricles can start to beat very fast (ventricular tachycardia) or quiver in an uncontrolled fashion (ventricular fibrillation).
In both
ventricular tachycardia, (V Tach) and ventricular fibrillation (V Fib), the heart can not do it's job properly. The ventricles can't fill with fresh blood from the lungs or pump the fresh blood to the body. That is why these abnormal heart rhythms can be deadly.
A cardiac defibrillator "depolarizes'' the heart muscles by delivery a pulse of electrical energy. This allows the rhythm to be reset by the sinus node and restore normal sinus rhythm.
There are two main types of cardiac defibrillators:
1- An emergency external defibrillator that can be used in sudden life threatening situations, as when person experiences an unexpected cardiac event
such as a heart attack. Emergency defibrillators are becoming increasing user-friendly and compact. Emergency defibrillators are often found on planes, airports and other public places, but are small enough so that they can be purchased by the public.
2- Implantable defibrillators are small devices inserted under the skin in high risk patients. These Implantable Cardioverter Defibrillators (ICD) are employed in patients at high risk for developing V Tach or V Fib. ICDs are capable of detecting the abnormal heart rate and delivery the electrical impulse to restore normal heart rhythm.
Updates on Cardiac Defibrillators
Luis Nombela-Franco, Cristina Daniela Mitroi, Ignacio Fernández Lozano, Arturo García-Touchard, Jorge Toquero Ramos, Victor Castro Urda, Jose Antonio Fernández Diaz, Elena Perez Pereira, Paula Beltrán Correas, Javier Segovia, Gerald S. Werner, Javier Goicolea Ruigomez, and Luis Alonso Pulpón
Circ Arrhythm Electrophysiol. 2011;CIRCEP.111.968008
published online before print December 28 2011
doi:10.1161/CIRCEP.111.968008
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JAMA Press Release
Many Patients With Implantable Cardioverter-Defibrillators Do Not Meet Criteria For Use
CHICAGO— study that included more than 100,000 patients who received implantable cardioverter-defibrillators (ICDs) found that about 20 percent did not meet evidence-based guidelines for receipt of an ICD, and that these patients had a significantly higher risk of in-hospital death than individuals who met criteria for receiving an ICD, according to a study in the January 5 issue of JAMA.
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LE KY, Sohail MR, Friedman PA, Uslan DZ, Cha SS, Hayes DL, Wilson WR, Steckelberg JM, Baddour LM; for the Mayo Cardiovascular Infections Study Group.
Pacing Clin Electrophysiol. 2011 Jan 5. doi: 10.1111/j.1540-8159.2010.02991.
Mayo School of Graduate Medical Education, Mayo Clinic, Rochester, Minnesota Divisions of Infectious Diseases Cardiovascular Diseases, Department of Medicine, Mayo Clinic, Rochester, Minnesota Division of Infectious Diseases, Department of Medicine, David Geffen School of Medicine, University of California Los Angeles, California Division of Biostatistics and Informatics, Mayo Clinic, Rochester, Minnesota.
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Featured Angina| Acute Coronary Syndrome and Heart Disease Interviews
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Author Interview: Claes Held MD
Associate professor at Uppsala Clinical Research Center and the
Cardiology department at Uppsala University Hospital in Sweden
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Publication:
Physical activity levels, ownership of goods promoting sedentary behaviour and risk of myocardial infarction: results of the INTERHEART study
Eur Heart J first published online January 11, 2012 doi:10.1093/eurheartj/ehr432
2012 doi:10.1093/eurheartj/ehr432
Claes Held, Romaina Iqbal, Scott A. Lear, Annika Rosengren, Shofiqul Islam,James Mathew, and Salim Yusuf
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What are the main findings of the study?
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The main findings are he following:
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It is well known that physical inactivity is a risk factor for developing cardiovascular disease.
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Our study shows that being physically active reduces the risk of having a heart attack.
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Older studies have mostly studied people in the developed countries. This study which has a global perspective and includes 52 countries from all continents, shows that physical inactivity reduces the risk also in both low- middle-and high-income countries.
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We also found that ownership of a car and TV was associated with a more than doubled risk of being sedentary and that in low- and middle income the risk for a heart attack was increased with 27 %.
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We can however, not conclude from this study that there is a causal relationship but it is an interesting finding.
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Were any of the findings unexpected?
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We looked at the relationship between physical activity both at work and during leisure time.
A bit surprising was that people with heavy physical labor did not have a reduction in the risk of heart attacks, whereas people with both mild and moderate intensity did.
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What should clinicians and patients take away from this study?
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The main findings above and also that physical activity with duration below the recommended 30 minutes/day does seem to prevent from heart attacks as well although not as much as when you do it according to guidelines.
This may be a comfort to those who are completely sedentary and feel that it would be a too big step to move to the 30 minutes/day right away.
All PA does a good job for the heart as compared to not doing anything at all!
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What recommendations do you have for cardiology health care providers as a result of your study?
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It would be interesting to follow up on the ownership of car and TV and do a prospective study to see if the theory holds true.
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Publication:
Short Telomere Length, Myocardial Infarction, Ischemic Heart Disease, and Early Death
- Maren Weischer, Stig E. Bojesen, Richard M. Cawthon, Jacob J. Freiberg,
Anne Tybjærg-Hansen, and Børge G. Nordestgaard
Arterioscler Thromb Vasc Biol. 2011;ATVBAHA.111.237271published online before print December 22 2011, doi:10.1161/ATVBAHA.111.237271 |
What are the main findings of the study?
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One in four Danes have reduced telomere length, that is, increased cellular ageing, and a 25% increased risk of early death and a 50% increased risk of heart attack.
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Were any of the findings unexpected?
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Yes, previous smaller studies have suggested much larger risk increases for early death and heart attack, while we found more modest risk estimates.
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What should clinicians and patients take away from this study?
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A possible prospect of the study is a simple blood test done by the general practitioner, which will reveal a person’s telomeric length and thereby the cellular wear and age.
If the celluar wear is more than expected by age alone, this could be a "wake-up call" suggesting for the patient that now is maybe the time to stop smoking and lose weight.
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What recommendations do you have for cardiology health care providers as a result of your study?
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We need to know if smoking and obesity is the direct cause of reduced telomeric length and thereby increased cellular wear, and whether decreased telomeric length is a direct cause of heart attack and early death.
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Angina | Heart Disease Resourses
| Chest Pain
Amazon.com 's Editorial Reviews
Angina: New Ways to Treat
Chronic Chest Pain
Part of the award winning public television series Healthy head
/Healthy Mind. It's one of the scariest medical symptoms for people who are generally healthy: a tightening, painful feeling in the chest known as Angina. In some cases this chest pain can be a serious warning that requires immediate treatment. But for the millions of people with chronic, stable angina the discomfort is something that can be readily managed with a variety of treatments. In this program we take a look at what generally causes angina, what can be done to prevent it and how it can be effectively treated with lifestyle changes, innovative drug therapies and medical procedures.
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Amazon.com Editorial Review:
Philips HeartStart Home Defibrillator (AED)
Be prepared for the unexpected.
When sudden cardiac arrest (SCA) strikes, the electrical system of the heart short circuits, causing the heart to quiver rather than pump in a normal rhythm. It typically results in the abnormal heart rhythm know as ventricular fibrillation (VF). It usually happens without warning and the majority of people have no previously recognized symptoms of heart disease. And it most often happens at home. For the best chance of survival from SCA caused by VF, a defibrillator should be used within 5 minutes. Yet, less than 1 in 20 people survive largely because a defibrillator does not arrive in time.
Just as seat belts or airbags do not save every life in a traffic accident, a defibrillator will not save every person who suffers a sudden cardiac arrest. Yet many lives could be saved if more people could be reached more quickly.
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